Research in recent years has highlighted that vitamin K deficiency, which is very common among Western people, is one of the major causes of cardiovascular and soft tissue calcification.
It has long been thought, and still is learned by everyone in school, that human gut bacteria produce enough vitamin K. Today, this has become doubtful, with measurements not confirming that a significant amount of vitamin K produced by intestinal bacteria would be produced and absorbed. Calcification of blood vessels and soft tissues is now practically considered an age-related lesion, although to the best of our knowledge, it is mainly caused by a lack of vitamin K. It follows, however, that vitamin K deficiency is considered to be practically general.
Analyzing the cardiovascular effects of vitamin K, it has now become clear that adequate levels of vitamin K intake can slow or even reverse the calcification of blood vessels and the heart . Based on several studies, vitamin K1 appears to be ineffective in this regard, while vitamin K2 is effective (e.g., Beulens et al., 2009). This is likely due to the fact that vitamin K2 raises blood levels more permanently and is otherwise more effective (Schurgers et al., 2007). In the 7-year follow-up Rotterdam study, among those with the highest vitamin K2 levels, there was a 60% reduction in heart mortality, a 26% reduction in all-cause mortality, and a 52% reduction in severe aortic calcification (Geleijnse et al., 2004). .
The role of vitamin K in osteoporosis: Osteoporosis is usually caused by low calcium intake and increased calcium intake in people with osteoporosis. However, this results in increased soft tissue and cardiovascular calcification. In recent years, several studies have shown that increased calcium supplementation, with or without vitamin D, increases the risk of cardiovascular death by 10-20%. Strangely, medical research lacks synthesizing minds that can simultaneously see the one-way results of osteoporosis, vitamin D research, and vitamin K research. Researchers always deal only with their own narrow field.
Vitamin K research has shown that osteocalcin, a protein associated with vitamin K, is found in large amounts in bone tissue. Because it is produced by bone cells, high levels are a sign of bone building. However, osteocalcin can only bind minerals and build them into bones if it is activated by vitamin K. Numerous studies have shown that low levels of vitamins K1 and K2 result in decreased bone density and more frequent pelvic, vertebral and femoral neck fractures. In a Japanese study e.g. those with the lowest vitamin K blood levels had a 3.6-fold higher risk of vertebral fracture than those with the highest vitamin K blood levels (Tsugawa et al., 2008). In Japan, menaquinone-4 (a type of vitamin K2) has long been used daily. This dose is 450 times the current recommendation.The treatment reduces fractures and strengthens the bone.
Vitamin K appears to be the next vitamin to play a vital role in preventing cardiovascular disease and osteoporosis.
Flavin7 bioflavonoid complex (red grape seed-husk drying, sorghum seed-husk drying, blackberry seed husk drying, black cherry husk drying, blackcurrant seed husk drying, redcurrant seed husk drying, plum husk drying, plum husk, plum husk) -vitamin (menaquinone-7), gelatin (capsule cover).
Recommended daily intake:
– 1 capsule.
Active ingredient in daily dose:
|Vitamin K2 (menaquinone-7)||100|
* g * NRV 133.3%
|16.3 mg + – 10%|
* NRV is% of recommended daily intake for adults.
– 36g (60 x 600 mg)
– 60 capsules.